Renal infarction

Renal infarction is called a rare urological condition in which the kidney tissue death (necrosis) due  Renal infarction occurs as a result of destruction of kidney tissue
 a complete and abrupt termination of large renal arterial blood flow. The disease mainly occurs in people of mature and elderly.

In contrast, in newborns often observed urate renal infarction, kidney wherein a large number of urate crystals are formed. Urate renal infarction in infants in most cases does not require treatment and resolves on its own.

Causes

Renal infarction often develops as a result of occlusion of the renal vessels emboli originating mural thrombus is normally the left ventricle or atrium. Therefore, renal infarction occurs frequently as one of various complications of cardiovascular disease:

  • Atrial fibrillation;
  • Infective endocarditis;
  • Atherosclerosis;
  • Mitral valve;
  • Periarteritis nodosa;
  • Myocardial infarction.

Also, renal infarction may develop in patients undergoing surgery for renal artery thrombosis or ascending aorta.

Urate renal infarction occurs in infants under the influence of birth stress. Very rarely urate renal infarction may be observed in adult patients suffering from gout, leukemia, extensive purulent or neoplastic processes in which there is a significant decay of tissue.

Renal infarction: the symptoms

The clinical picture of myocardial kidney depends on the amount of area of ​​necrosis of renal tissue. With low renal infarction symptoms may be entirely absent. Large renal infarction appear sharp, severe pain in the lumbar region, the appearance of blood in the urine impurities (hematuria), in some patients there is a decrease in urine output. Starting from 2 - 3 days from the onset of the disease in patients with increased body temperature (37 0 - 37, 5), observed arterial hypertension, occurring due to ischemia of the tissues surrounding the area of ​​necrosis. Other symptoms of a heart attack the kidneys are severe nausea and vomiting.

If urate renal infarction observed clouding of urine and change its color to the acquisition of brown. In diapers and diaper stains form a characteristic reddish-brown color. The same light precipitate can be seen in and around the urethral opening. In the analysis of urine when kidney urate infarction revealed a high content of urate (ammonium and sodium salts of uric acid).

Treatment of heart attack kidney

At  The clinical picture of renal infarction
 renal infarction patients prescribed strict bed rest. To prevent re-blockage of the renal arteries and further thrombus administered anticoagulants (which reduce blood clotting). During the first three hours of the onset of illness justified by the use of thrombolytic drugs that can dissolve the clot, a sprig of renal artery clog.

To restore normal renal blood flow in certain cases shown holding balloon angioplasty. Through the femoral artery, the doctor inserts a special catheter with a balloon at the end. Then, it is advanced under X-ray control in renal artery stoppered. After the doctor make sure the correct position of the catheter, it inflates the balloon, thereby crushing it clogs the blood clot. This operation is performed in the first hours of the disease, is not yet developed necrosis, renal tissue, and there is a high likelihood of recovery of its functions.

But the treatment of renal urate infarction, despite the intimidating name of the disease is not carried out. Newborn urate kidney infarction refers to the so-called transient (transient) states and is one of the physiological norm. The child is given excessive drinking, which leads to an increase in urine output from the kidneys and wash out the crystals of uric acid salts. As a result, the symptoms of heart attack kidney urate gradually disappear, and disappear completely in 10 - 15 days of age of the baby.





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